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Text Version   RSS   Subscribe   Unsubscribe   Archive   Media Kit          January 13, 2015

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ASSOCIATION NEWS

Now is the time to renew your 2015 ASHI Membership online

Doing so today so will help to avoid interruption accessing the ASHI Quarterly & ASHI-U for continuing education credits. Your Human Immunology subscription can also be renewed at this time.
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In 2014, ASHI celebrated it's 40th anniversary!
The study of human histocompatibility has grown from a few modest research efforts studying human genetics in the 1950s to a broadly based science merging immunology and genetics, including transplantation of organs and tissues, susceptibility to disease, regulation of immune responsiveness and molecular characterization of the unique supergene HLA. Click here for a brief history of ASHI’s beginnings.

Watch the video commemorating ASHI’s 40th anniversary.

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2015 ASHI Regional Education Workshops

2015 ASHI Regional Education Workshops
Workshop 1: San Antonio, Texas — Thursday, April 30 - Saturday, May 2

Workshop 2: Philadelphia, Pennsylvania — Thursday, June 19 - Saturday, June 21

For more information on the ASHI Regional Education Workshops, please visit the Regional Meetings page.

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Welcome to ASHI U!

As part of our mission, ASHI strives to be foremost authority and leading educational resource in immunogenetics and histocompatibility. The ASHI University curriculum is a valuable resource to technicians, technologists, directors, clinicians, coordinators and educators.

As a Society, we embrace the opportunity to provide education to our members. ASHI U is a great way to earn Continuing Education Credits online by reading the content in the modules and taking the quizzes. ASHI members have free access to modules and quizzes!


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In Memoriam

Dear Colleagues,

Please join us in remembering those who have recently passed.

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INDUSTRY NEWS


HLA-G and susceptibility to develop celiac disease
Human Immunology
The Human Leukocyte Antigen-G has immunomodulatory function and its expression has been associated with several diseases. In this study scientists analyzed HLA-G polymorphisms in order to evaluate their possible association with susceptibility to celiac disease development. A total of 420 celiac patients and 509 controls were genotyped for HLA-G polymorphisms. They sequenced 800 bp upstream the ATG codon and the whole 3′ untranslated region of the HLA-G gene, whereas the ΔC deletion at exon 3 was detected by RFLP-PCR.
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Increased expression of human leucocyte antigen class I free heavy chains on monocytes of patients with spondyloarthritis and cells transfected with HLA-B27
International Journal of Immunogenetics
Human leucocyte antigen (HLA)-B27 expression is correlated with spondyloarthritis (SpA), but its role in disease pathogenesis remains unclear. The aim of the study was to determine whether HLA-B27 free heavy chain (FHC) contributes to SpA pathogenesis. Flow cytometry was used to analyse the FHC expression on CD3+ and CD14+ cells in the peripheral blood and synovial fluid from SpA patients, healthy controls and rheumatoid arthritis patients.
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HLA dosage effect in narcolepsy with cataplexy
Immunogenetics
Narcolepsy with cataplexy is a sleep disorder caused by the loss of hypocretin-producing neurons in the hypothalamus. It is tightly associated with a specific human leukocyte antigen (HLA)-allele: HLA-DQB1*06:02. Based on this, an autoimmune process has been hypothesized. A functional HLA-DQ molecule consists of a DQα and a DQβ chain. HLA-DQB1*06:02 (DQβ) has a strong preference for binding to HLA-DQA1*01:02 (DQα), and together they form the functional DQ0602 dimer.
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ASHI Insights
Colby Horton, Vice President of Publishing, 469.420.2601
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Lauren Swan, Content Editor, 202.684.7496  
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