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The 41th Annual Meeting website is now live!
Join us in Savannah, Georgia, Sept. 28-Oct. 2, 2015 at the Savannah International Trade & Convention Center. This year's annual meeting draws inspiration from its location by striving to provide attendees with a little “southern hospitality” and a program built with something for everyone. Visit the annual meeting website for the latest information on this year's meeting including the full program, event information, Savannah hot spots, and more. Explore and make plans to attend this year's meeting today!
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ASHI co-sponsored symposia

FOCIS is a translational immunology meeting delivering the latest breakthroughs across immune-mediated diseases. ASHI is co-sponsoring two symposia at FOCIS this year. Click here to read more and then plan on attending!

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2015 board election and proposed bylaws amendment ballot
Ballots were mailed out to full-doctoral and non-doctoral ASHI members last week. Click here to view the nominees.


The following guidelines must be followed in order for these ballots to be valid.
  1. Do not write your name on this ballot.
  2. Print and sign your name on the inside flap of the pre-addressed return envelope. If the envelope is received without this information, the ballot will be invalid.
  3. Place your completed ballot in the pre-addressed return envelope, seal and mail.
  4. To be counted, your ballot must be postmarked by August 3, 2015. Ballots postmarked after that date will be invalid.
  5. Only the original ballot will be accepted for tabulation of votes. Ballots that have been copied or altered in any way will not be counted.

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Call for volunteers
Please let us know if you would like to become a member of an ASHI Committee/Program. Place a mark next to each committee/program in which you are interested and indicate your preference (i.e. 1st, 2nd, 3rd). Completed forms will be used to identify volunteers to fill openings on various committees for 2016.
To be considered, please fill out the volunteer form and return your completed form to the ASHI Staff Office via fax (856-439-0525) or e-mail (

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ASHI Career Center has been updated!

Check out the newer, user friendly ASHI Career Center.

The site is now easier to post and search for jobs and has been optimized for mobile devices so you can use your smartphone and tablet to post and search for jobs! Create a Job Alert to let jobs find you wherever you are! Search for jobs using keywords or title by state.

Employers — View products/pricing

Job Seekers — Search jobs and post your resume

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Introducing a new manuscript format: Enabling access to immunogenomic population data with short population reports
Human Immunology
The capacity to replicate the findings of a study is key to the advancement of research, and access to the data on which a study is predicated is required for true replication. Population genetic studies have long been a focus of the immunogenetic research community, but access to the primary genotype data underlying these studies has historically been limited. With the notable exception of the International HLA and Immunogenetics Workshops, it is primarily allele and haplotype frequency data that are made available upon publication for most immunogenetic population studies.
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Functional and genetic diversity of leukocyte immunoglobulin-like receptor and implication for disease associations
Journal of Human Genetics
Human leukocyte immunoglobulin-like receptors (LILR) are a family of 11 functional genes encoding five activating (LILRA1, 2, 4–6), five inhibitory (LILRB1–5) and one soluble (LILRA3) form. The number of LILR genes is conserved among individuals, except for LILRA3 and LILRA6, which exhibit copy-number variations. The LILR genes are rapidly evolving and showing large interspecies differences, making it difficult to analyze the functions of LILR using an animal model. LILRs are expressed on various cells such as lymphoid and myeloid cells and the expression patterns are different from gene to gene.
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Construction of a population-specific HLA imputation reference panel and its application to Graves' disease risk in Japanese
Nature Genetics
To fine map association signals of human leukocyte antigen (HLA) variants in the major histocompatibility complex (MHC) region, we constructed a Japanese population-specific reference panel (n = 908). We conducted trans-ancestry comparisons of linkage disequilibrium (LD) and haplotype structure for HLA variants using an entropy-based LD measurement, ε, and a visualization tool to capture high-dimensional variables. Our Japanese reference panel exhibited stronger LD between HLA genes than European or other East Asian populations, characterized by one population-specific common long-range HLA haplotype.
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ASHI Insights
Colby Horton, Vice President of Publishing, 469.420.2601
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